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It alleviates hepatic injury by activating the Nrf2/HO-1 antioxidant pathway and suppressing NF-Bmediated inflammation, highlighting its potential as a therapeutic applicant for ALD (94)
Data from participants without events of interest were censored at the end of their in-trial period
demonstrated improvements in endothelial function markers in older adults following glutathione repletion [10][21]
Finally, transfected neurons were immunostained for presynaptic markers, VGLUT1 and synapsin, and postsynaptic marker, PSD-95, to assess functional synapse formation
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