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This age-related decline has made it a subject of considerable interest in longevity and tissue repair research
17 While ERAD was previously thought to be associated with PQC, which safeguards protein maturation, recent studies have demonstrated that ERAD also regulates the abundance of folding-competent proteins as a means to fine-tune key physiological processes, suggesting that ERAD constitutively functions to ensure both protein fidelity and abundance
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Our results indicated that NAC effectively promoted the differentiation of TSPCs and the repair of injured tendons in rats
