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These adaptions drive tumor proliferation, survival, and migration by fundamentally reshaping cellular biological characteristics [4]
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(2) developing combination therapies for specific pathological subtypes, including potential synergistic benefits with mitochondria-targeted antioxidants (e.g., MitoQ) or bile acid modulators (e.g., ursodeoxycholic acid, UDCA) in MASH patients with prominent ballooning degeneration or cholestatic features
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