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327 In the inflammatory environment, the M1 to M2 shift promoted by GLP-1 is crucial for reducing the expression of inflammatory factors such as IL-6, TNF-, and iNOS

This suggests that Epitalon may not just restore telomere length through enzymatic activation, but it may actually reprogram the transcriptional readiness of cells, shifting their expression profiles toward a more youthful state

Specifically, drugs that target inflammation pathways unique to the heart, such as those modulating inflammatory responses associated with myocardial fibrosis or cardiomyocyte (200)

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