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[7] implicated 4-OH-E 2 in the induction of estrogen-responsive genes, a response that exhibited partial or no abrogation by coadministration with an antiestrogen, providing evidence for the ability of 4-OH-E 2 to carry out genetic upregulation via a pathway independent of ER signalling
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Gut 54 : 16931698 Acknowledgements R Nogueiras is a recipient of a Marie Curie Outgoing International Fellowship
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