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ERR biology, coactivation, and the exercise-mimetic concept ERRs operate as transcription factors that bind response elements in promoters and enhancers of metabolic genes, recruiting coactivators such as PGC-1/ and chromatin-remodeling complexes

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Research has shown increased phosphorylation of extracellular signal-regulated kinases (ERK) 1 and 2, along with their downstream targets, including c-Fos, c-Jun, and Egr-1, which are molecules involved in cell growth, migration, and angiogenesis