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This reaction causes the production of unstable molecules called free radicals

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(131) proved the neuroprotective efficacy of intracerebroventricular injection (i.c.v.) of Ex-4 exerted after transient ischemia/reperfusion in rats and first suggested that Ex-4 played a neuroprotective role probably by stimulating the GLP-1R/-endorphin signal pathway in hippocampal microglia (76)

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