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N-acetylcysteine (NAC), a nephroprotective agent, maintains mitochondrial redox homeostasis by enhancing mitochondrial GSH activity
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Given that GABA functions to reduce neuronal excitability in the CNS, the NHPN-1010induced enhancement of 1 subunit expression in the DCN could indicate a reversal of central neural hyperactivity
The more practical question is whether peptides can complement existing therapies and potentially allow reduction of treatments that cause long-term side effects
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