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In metabolic dysfunctionassociated steatohepatitis (MASH), for example, it remains uncertain whether improvements in hepatic inflammation and fibrosis are exclusively secondary to weight loss and metabolic improvements, or whether direct hepatoprotective mechanisms are involved
Those are the exact same molecules that drive psoriatic arthritis, rheumatoid arthritis, and lupus
These agents have both hormonal and brain targets
if you are still in the titration phase or on a subtherapeutic maintenance dose, increasing to the next level often restarts weight loss
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