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IP 3 Rs mediate the Ca spike response in the apical cytoplasm triggered by physiological concentrations of the neurotransmitter acetylcholine, whereas RyRs are activated by nicotinic acid adenine dinucleotide phosphate and cyclic ADP-ribose in response to physiological levels of cholecystokinin

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However, like all research peptides, it must be used only in non-human laboratory settings , and should never be promoted or interpreted as a therapeutic product