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Restricting -synuclein transport into mitochondria by inhibition of -synucleinVDAC complexation as a potential therapeutic target for parkinsons disease treatment
Our study showed that HepG2 cells, which primarily rely on extracellular cystine as a source of cysteine 20 , responded to cystine deprivation by reducing anabolic signaling including attenuation of mTORC1 pathway and activation of the ISR (Fig
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Analysis of enhanced substrate response to glutathione in dairy products To evaluate the detection performance of AgNPs enhanced substrates in practical applications, the determination of glutathione in dairy products was investigated
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