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Meanwhile, in January 1996 the first rodent study suggested that GLP-1 could be a central regulator of feeding behavior and satiety based on intracerebroventricular injection studies [4], but soon thereafter the results of other rodent studies using peripherally injected GLP-1 argued against an appetite effect [5]

That comes from the way it works: hitting three hormone pathways at once (GLP-1, GIP, and glucagon) to control appetite, support insulin, and burn fat

Para obtener ms informacin sobre los agonistas de GLP-1, consulte Actualizaciones de anestesia en NYSORA Aplicacin del manual de anestesia

1 2 The Lancet