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The disconnect between short plasma half-life and prolonged therapeutic effects observed in many studies supports tissue-level mechanisms extending beyond circulating peptide concentrations
In cases where back pain is not the direct result of an injury, it is usually caused by inflammation
The preclinical literature has described effects across multiple pathways, and mechanistic claims are better framed as a family of related observations rather than a settled mechanism
It helps convert fats and proteins into energy, boosts metabolism, and supports the body's natural detoxification processes
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