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Nephroprotective effect of apilarnil in lipopolysaccharide-induced sepsis through TLR4/NF-B signaling pathway
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The peptide is most commonly injected near the site of injury or in areas requiring therapeutic intervention
It inhibits NOTCH1 signaling by using its GCT activity to cleave a -glutamyl-cysteine bond at glutamate 1669 of the immature NOTCH receptor, thereby preventing the essential S1 cleavage and subsequent activation ( FIGURE 6 The physiological importance of this mechanism is underscored by severe developmental phenotypes upon its disruption
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